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New Therapeutic Approach for Intestinal Fibrosis Through Inhibition of pH-Sensing Receptor GPR4
UploadHypoxia Reduces the Transcription of Fibrotic Markers in the Intestinal Mucosa
Download from doi.orgA Novel OGR1 (GPR68) Inhibitor Attenuates Inflammation in Murine Models of Colitis
Download from doi.orgThe Oxysterol Synthesising Enzyme CH25H Contributes to the Development of Intestinal Fibrosis
UploadSuccinate receptor mediates intestinal inflammation and fibrosis
UploadIntestinal activation of pH-sensing receptor OGR1 (GPR68) contributes to fibrogenesis
UploadGp96 deficiency affects TLR4 functionality and impairs ERK and p38 phosphorylation
Download from doi.orgThe proton-activated receptor GPR4 modulates intestinal inflammation
Download from doi.orgFactors Promoting Development of Fibrosis in Crohn’s Disease
Download from doi.orgTitanium dioxide nanoparticles exacerbate DSS-induced colitis: role of the NLRP3 inflammasome
Download from gut.bmj.comSialic acid catabolism drives intestinal inflammation and microbial dysbiosis in mice
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