Cambridge University Press, Behavioral and Brain Sciences, 03(31)
DOI: 10.1017/s0140525x08004421
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AbstractCrespi & Badcock (C&B) have presented a novel view that the influence of genomic imprinting causes diametrically opposite disorders: namely, psychoses and autism. I propose an extended hypothesis that while genomic imprinting is likely to have an influence on the pathogenesis of psychoses and autism, it might contribute to phenomenological antithesis between as well as within these disorders.