260 papers found
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Enteropathogenic Escherichia coli and Vaccinia Virus Do Not Require the Family of WASP-Interacting Proteins for Pathogen-Induced Actin Assembly
The Age of Gene Discovery in Very Early Onset Inflammatory Bowel Disease
The actin regulator N-WASp is required for muscle-cell fusion in mice
Wiskott-Aldrich Syndrome Protein Deficiency in Innate Immune Cells Leads to Mucosal Immune Dysregulation and Colitis in Mice
Tu1968 Increased Sensitivity to TGF Results in Defects in T Cell Proliferation and Regulatory T Cell Induction in the Absence of Wiskott-Aldrich Syndrome Protein (WASp)
Mo1798 Neural Wiskott-Aldrich Syndrome Protein (N-WASp) Regulates Tight Junction Integrity and is Required for EPEC-Mediated Epithelial Barrier Disruption
993 WASp Deficiency in Innate Immune Cells Leads to Severe Colitis Associated With Impairment in IL-10 Production
Wiskott-Aldrich syndrome protein (WASP) and N-WASP are critical for peripheral B-cell development and function
Update on biologic pathways in inflammatory bowel disease and their therapeutic relevance
Dysfunction of the intestinal microbiome in inflammatory bowel disease and treatment
Colitis in mice with WASP-Deficient myleoid cells is associated with defects in IL-10 secretion and can be rescued with exogenous IL-10: P-211.
Immunobiology of T Cells in Inflammatory Bowel Disease
Gut-tropic T Cells that Express Integrin α4β7 and CCR9 are Required for Induction of Oral Immune Tolerance in Mice
Rac1 drives melanoblast organization during mouse development by orchestrating pseudopod-driven motility and cell cycle progression
Decoding inflammatory bowel disease through the lens of immunodeficiency
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