227 papers found
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In situ cytokine therapy: redistribution of clonally expanded T cells
Induction of systemic CTL responses in melanoma patients by dendritic cell vaccination: Cessation of CTL responses is associated with disease progression
thor Straten, P. et al. In situ cytokine therapy: redistribution of clonally expanded T cells. Eur. J. Immunol. 31, 250-258
Quantification of melanoma cell-specific MART-1 mRNA in peripheral blood by a calibrated competitive reverse transcription-PCR.
Differential expression of CD28 and CD94 / NKG2 on T cells with identical TCR beta variable regions in primary melanoma and sentinel lymph node
Aberrant p27(Kip1) promoter methylation in malignant melanoma
Comparative delineation of T cell clonotypes in coexisting syngeneic B16 melanoma
Tumor Infiltrating Lymphocytes in Melanoma Comprise High Numbers of T-Cell Clonotypes That Are Lost during in Vitro Culture
CD40-ligated dendritic cells effectively expand melanoma-specific CD8+ CTLs and CD4+ IFN-γ-producing T cells from tumor-infiltrating lymphocytes
Expression of CD94/NKG2 Subtypes on Tumor-Infiltrating Lymphocytes in Primary and Metastatic Melanoma
Anti-Melanocyte T Cell Responses – Methodology Versus Biology
T-cell clonality in immune responses
Accumulation of Identical T Cells in Melanoma and Vitiligo-Like Leukoderma
Function and dysfunction of CD4 + T cells in the immune response to melanoma
In situ T cells in melanoma
Kirkin AF, Thor Straten P, Hansen MR, Barfoed A, Dzhandzhugazyan KN, Zeuthen JEstablishment of gp100 and MART-1/Melan-A-specific cytotoxic T lymphocyte clones using in vitro immunization against preselected highly immunogenic melanoma cell clones. Cancer Immunol Immunother 48: 239-246
In Situ T Cell Responses Against Melanoma Comprise High Numbers of Locally Expanded T Cell Clonotypes1
Somatic mutation of the Peutz-Jeghers syndrome gene, LKB1/STK11, in malignant melanoma
Somatic Fas Mutations in Non-Hodgkin’s Lymphoma: Association With Extranodal Disease and Autoimmunity
Activation of preexisting T cell clones by targeted interleukin 2 therapy
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