Lina Schiffer
0000-0001-8540-4861
University of Miami
24 papers found
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Inhibition of the glucocorticoid‐activating enzyme 11β‐hydroxysteroid dehydrogenase type 1 drives concurrent 11‐oxygenated androgen excess
Tissue Glucocorticoid Metabolism in Adrenal Insufficiency: A Prospective Study of Dual-release Hydrocortisone Therapy
Classic and 11-oxygenated androgens in serum and saliva across adulthood: a cross-sectional study analyzing the impact of age, body mass index, and diurnal and menstrual cycle variation
Multi-steroid profiling by UHPLC-MS/MS with post-column infusion of ammonium fluoride
Multi-steroid profiling by uPLC-MS/MS with post-column infusion of ammonium fluoride
Peripheral blood mononuclear cells preferentially activate 11-oxygenated androgens
11-oxygenated estrogens are a novel class of human estrogens but do not contribute to the circulating estrogen pool
Intracrine Testosterone Activation in Human Pancreatic β Cells Stimulates Insulin Secretion.
The A-ring reduction of 11-ketotestosterone is efficiently catalysed by AKR1D1 and SRD5A2 but not SRD5A1
Testosterone enhances GLP-1 efficacy at the plasma membrane and endosomes to augment insulin secretion in male pancreatic β cells
Human steroid biosynthesis, metabolism and excretion are differentially reflected by serum and urine steroid metabolomes: A comprehensive review
Measurement of Salivary Adrenal-Specific Androgens as Biomarkers of Therapy Control in 21-Hydroxylase Deficiency
Steroid Metabolome Analysis in Disorders of Adrenal Steroid Biosynthesis and Metabolism
A liquid chromatography-tandem mass spectrometry assay for the profiling of classical and 11-oxygenated androgens in saliva
Understanding the Role of Androgen Action in Female Adipose Tissue
A novel high-throughput assay for the measurement of salivary progesterone by liquid chromatography tandem mass spectrometry
Intracrine androgen biosynthesis, metabolism and action revisited
MECHANISMS IN ENDOCRINOLOGY: The sexually dimorphic role of androgens in human metabolic disease
The steroid metabolite 16(β)-OH-androstenedione generated by CYP21A2 serves as a substrate for CYP19A1
Biotransformation of the mineralocorticoid receptor antagonists spironolactone and canrenone by human CYP11B1 and CYP11B2: Characterization of the products and their influence on mineralocorticoid receptor transactivation
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