Yousuke Takahama
0000-0002-4992-9174
National Institutes of Health
246 papers found
Refreshing results…
Cluster formation among small resting B lymphocytes leading to B cell activation
Different Effect of Prostaglandin-E2 on B-Cell Activation by 2 Distinct B-Cell Differentiation Factors, B151-Trf1/Il-5 and B151-Trf2 - Selective-Inhibition of B151-Trf2-Induced Antibody-Response Through Increases in Intracellular Cyclic-Amp Levelsl
Identification of a B cell subset responsive to a T cell-derived lymphokine B151-TRF2 by monoclonal antibody specific for N-acetylglucosamine residue(S)
Identification of autoantibody-inductive B cell-stimulation factor and gene regulation of an MHC-linked B cell response
The Activation of L3T4+ Helper T Cells Assisting the Generation of Anti-Tumor Lyt-2+ Cytotoxic T Lymphocytes: Requirement of 1a-Positive Antigen-Presenting Cells for Processing and Presentation of Tumor Antigens
Ia-restricted B-B cell interaction. I. The MHC haplotype of bone marrow cells present during B cell ontogeny dictates the self-recognition specificity of B cells in the polyclonal B cell activation by a B cell differentiation factor, B151-TRF2.
Polyclonal B cell activation by a B cell differentiation factor, B151-TRF2. III. B151-TRF2 as a B cell differentiation factor closely associated with autoimmune disease.
Polyclonal B-cell activation by a B-cell differentiation factor B151-TRF2. IV. B151-TRF2-responsive F1 B cells consist of two separate populations capable of recognizing only one of the parental I-A products expressed on B cells
Polyclonal B-Cell Activation by a B-Cell Differentiation Factor B151-Trf2 .4. B151-Trf2-Responsive F1 B-Cells Consist of 2 Separate Populations Capable of Recognizing Only One of the Parental I-a Products Expressed on B-Cells
Ia-Restricted B-B Cell-Interaction .1. The MHC Haplotype of Bone-Marrow Cells Present During B-Cell Ontogeny Dictates the Self-Recognition Specificity of B-Cells in the Polyclonal B-Cell Activation by a B-Cell Differentiation Factor, B151-Trf2
Polyclonal B-Cell Activation by a B-Cell Differentiation Factor, B151-Trf2 .3. B151-Trf2 as a B-Cell Differentiation Factor Closely Associated With Autoimmune-Disease
Polyclonal B cell activation by a B cell differentiation factor, B151-TRF2. II. Evidence for interaction of B151-TRF2 with glycoprotein on B cell membrane via recognition of terminal N-acetyl-D-glucosamine residue(s).
Polyclonal B cell activation by B cell differentiation factor B151-TRF2. I. Involvement of self-Ia recognition process mediated by B cells.
Identification of two distinct factors, B151-TRF1 and B151-TRF2, inducing differentiation of activated B cells and small resting B cells into antibody-producing cells.
Identification of 2 Distinct Factor-B151-Trf1 and Factor-B151-Trf2 Inducing Differentiation of Activated B-Cells and Small Resting B-Cells Into Antibody-Producing Cells
Polyclonal B-Cell Activation by a B-Cell Differentiation Factor, B151-Trf2 .2. Evidence for Interaction of B151-Trf2 With Glycoprotein on B-Cell Membrane via Recognition of Terminal N-Acetyl-D-Glucosamine Residue(s)
Polyclonal B-Cell Activation by B-Cell Differentiation Factor B151-Trf2
Role of N-acetyl-D-galactosamine residue on B151K12-derived T cell-replacing factor (B151-TRF) molecule in B cell-receptor binding and -stimulating activity.
B-Cell Growth and Differentiation Activity of a Purified T-Cell-Replacing Factor (Trf) Molecule From B151-T-Cell Hybridoma
Role of N-Acetyl-D-Galactosamine Residue on B151k12-Derived T-Cell-Replacing Factor (B151-Trf) Molecule in B-Cell-Receptor Binding and B-Cell-Stimulating Activity
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