Amit Singh
0000-0001-6761-1664
Indian Institute of Science
24 papers found
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Longitudinal single cell atlas identifies complex temporal relationship between type I interferon response and COVID-19 severity
Moxifloxacin-Mediated Killing of Mycobacterium tuberculosis Involves Respiratory Downshift, Reductive Stress, and Accumulation of Reactive Oxygen Species
S -Adenosylmethionine–responsive cystathionine β-synthase modulates sulfur metabolism and redox balance in Mycobacterium tuberculosis
Mycobactin Analogues with Excellent Pharmacokinetic Profile Demonstrate Potent Antitubercular Specific Activity and Exceptional Efflux Pump Inhibition
Glycolysis downregulation is a hallmark of HIV‐1 latency and sensitizes infected cells to oxidative stress
Efficacy of the β-lactam\β-lactamase inhibitor combination is linked to WhiB4 mediated changes in redox physiology of Mycobacterium tuberculosis
MUSASHI-mediated expression of JMJD3, a H3K27me3 demethylase, is involved in foamy macrophage generation during mycobacterial infection
Identifying and Tackling Emergent Vulnerability in Drug-Resistant Mycobacteria
Emerging Role and Characterization of Immunometabolism: Relevance to HIV Pathogenesis, Serious Non-AIDS Events, and a Cure
Glucose Metabolism in T Cells and Monocytes: New Perspectives in HIV Pathogenesis
Mycobacterium tuberculosis Inhibits RAB7 Recruitment to Selectively Modulate Autophagy Flux in Macrophages
Polyketide Quinones Are Alternate Intermediate Electron Carriers during Mycobacterial Respiration in Oxygen-Deficient Niches
Differential proteomics approach to identify putative protective antigens of Mycobacterium tuberculosis presented during early stages of macrophage infection and their evaluation as DNA vaccines
Analysis of the hierarchical structure of the B. subtilis transcriptional regulatory network
Measuring Glutathione Redox Potential of HIV-1-infected Macrophages*
Reengineering Redox Sensitive GFP to Measure Mycothiol Redox Potential of Mycobacterium tuberculosis during Infection
Detection of Membrane Potential in Mycobacterium tuberculosis
Mycobacterium Tuberculosis WhiB4 is a Redox – Dependent Nucleoid Associated Protein
Reductive Stress in Microbes: Implications for Understanding Mycobacterium tuberculosis Disease and Persistence
Mycobacterium tuberculosis WhiB3 Maintains Redox Homeostasis by Regulating Virulence Lipid Anabolism to Modulate Macrophage Response
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