Caitlin Carroll
0000-0003-1572-7360
Wake Forest University
11 papers found
Refreshing results…
Kir6.2-KATPchannels alter glycolytic flux to modulate cortical activity, arousal, and sleep-wake homeostasis
Upsetting the Balance: How Modifiable Risk Factors Contribute to the Progression of Alzheimer’s Disease
Novel method for collecting hippocampal interstitial fluid extracellular vesicles (EVISF) reveals sex‐dependent changes in microglial EV proteome in response to Aβ pathology
KATP channels are necessary for glucose-dependent increases in amyloid-β and Alzheimer’s disease–related pathology
Novel method for collecting hippocampal interstitial fluid extracellular vesicles (EV-ISF) reveals sex-dependent changes in microglial EV proteome in response to Aβ pathology
Acute hyper- and hypoglycemia uncouples the metabolic cooperation between glucose and lactate to disrupt sleep
Kir6.2-containing KATP channels are necessary for glucose dependent increases in amyloid-beta and Alzheimer’s-related pathology
Sulfonylureas target the neurovascular response to decrease Alzheimer’s pathology
The Interaction Between Sleep and Metabolism in Alzheimer’s Disease: Cause or Consequence of Disease?
Targeted Deletion of Hepatocyte Abca1 Increases Plasma HDL (High-Density Lipoprotein) Reverse Cholesterol Transport via the LDL (Low-Density Lipoprotein) Receptor
Cortical zeta-inhibitory peptide injection reduces local sleep need
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