Léa Lemaitre
0000-0003-1940-1018
Stanford Medicine
12 papers found
Refreshing results…
Molecular and immune landscape of hepatocellular carcinoma to guide therapeutic decision-making
Multiplatform single cell spatial dissection of the invasive front of hepatocellular carcinoma (HCC) reveals molecular insights into tumor progression
Data from MYC Overexpression Drives Immune Evasion in Hepatocellular Carcinoma That Is Reversible through Restoration of Proinflammatory Macrophages
Supplementary Tables from MYC Overexpression Drives Immune Evasion in Hepatocellular Carcinoma That Is Reversible through Restoration of Proinflammatory Macrophages
Supplementary Figures from MYC Overexpression Drives Immune Evasion in Hepatocellular Carcinoma That Is Reversible through Restoration of Proinflammatory Macrophages
MYC Overexpression Drives Immune Evasion in Hepatocellular Carcinoma That Is Reversible through Restoration of Proinflammatory Macrophages
Spatial Multi-Omic Immune Profiling and Functional Characterization of Hepatocellular Carcinoma (Hcc) Reveal Mechanisms of Minimal Residual Disease (Mrd)
Toll-like receptor 4 selective inhibition in medullar microenvironment alters multiple myeloma cell growth
Eomes-Dependent Loss of the Co-activating Receptor CD226 Restrains CD8+ T Cell Anti-tumor Functions and Limits the Efficacy of Cancer Immunotherapy
Circulating CD14highCD16lowintermediate blood monocytes as a biomarker of ascites immune status and ovarian cancer progression
Imprinting of Mesenchymal Stromal Cell Transcriptome Persists even after Treatment in Patients with Multiple Myeloma
Tumor cells educate mesenchymal stromal cells to release chemoprotective and immunomodulatory factors
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