Natalia Felipe-Medina
Instituto de Biología Molecular y Celular del Cáncer
17 papers found
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Table S5 from The Oncogenic FOXL2 C134W Mutation Is a Key Driver of Granulosa Cell Tumors
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Uploadtable s4 from The Oncogenic FOXL2 C134W Mutation Is a Key Driver of Granulosa Cell Tumors
Uploadtable s1 from The Oncogenic FOXL2 C134W Mutation Is a Key Driver of Granulosa Cell Tumors
UploadSupplementary Data from The Oncogenic FOXL2 C134W Mutation Is a Key Driver of Granulosa Cell Tumors
UploadData from The Oncogenic FOXL2 C134W Mutation Is a Key Driver of Granulosa Cell Tumors
UploadTable S2 from The Oncogenic FOXL2 C134W Mutation Is a Key Driver of Granulosa Cell Tumors
UploadA Human Hereditary Cardiomyopathy Shares a Genetic Substrate With Bicuspid Aortic Valve
UploadThe Oocyte-Specific Linker Histone H1FOO Is Not Essential for Mouse Oogenesis and Fertility
Download from doi.orgThe Oncogenic FOXL2 C134W Mutation Is a Key Driver of Granulosa Cell Tumors
Download from aacrjournals.orgA cryptic BRCA2 repeated motif binds to HSF2BP oligomers with no impact on meiotic recombination
UploadThe PSMA8 subunit of the spermatoproteasome is essential for proper meiotic exit and mouse fertility
Download from doi.orgC14ORF39/SIX6OS1 is a constituent of the synaptonemal complex and is essential for mouse fertility
Download from www.nature.comSororin loads to the synaptonemal complex central region independently of meiotic cohesin complexes
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