Valerie Crepel
www.inmed.fr
0000-0003-0408-3766
INSERM Délégation régionale Provence Alpes Cote d'Azur et Corse
64 papers found
Refreshing results…
Modulatory sites of NMDA receptors and regulation of long-term consequences of ischemia
A long-lasting calcium-activated nonselective cationic current is generated by synaptic stimulation or exogenous activation of group I metabotropic glutamate receptors in CA1 pyramidal neurons.
Blocking GABA(A) inhibition reveals AMPA- and NMDA-receptor-mediated polysynaptic responses in the CA1 region of the rat hippocampus.
Intracellular injection of a Ca2+ chelator prevents generation of anoxic LTP
Glutamate metabotropic receptors increase a Ca(2+)-activated nonspecific cationic current in CA1 hippocampal neurons
Anoxic LTP: A switch in favor of NMDA receptor-mediated synaptic transmission
A selective LTP of NMDA receptor-mediated currents induced by anoxia in CA1 hippocampal neurons
Anoxia-induced LTP of isolated NMDA receptor-mediated synaptic responses
The NMDA receptor contributes to anoxic aglcemic induced irreversible inhibition of synaptic transmission
The K+ channel opener diazoxide enhances glutamatergic currents and reduces GABAergic currents in hippocampal neurons
A Brief Anoxic-Aglycaemic Episode Induces a Persistent Enhancement of the Synaptic and Exogenous Nmda Currents, in Rat Hippocampal Slices
Sulfonylureas Reduce the Slowly Inactivating D-Type Outward Current in Rat Hippocampal-Neurons
A Novel Form of LTP Induced by Anoxia in Ca1 Hippocampal-Neurons
Cycloheximide Reduces the Effects of Anoxic Insult In Vivo and In Vitro
Developmental and regional differences in the vulnerability of rat hippocampal slices to lack of glucose
Glibenclamide depresses the slowly inactivating outward current (ID) in hippocampal neurons
Atp Sensitive K+ Channels and Anoxia
Transient cerebral ischemia induces changes in SRIF mRNA in the fascia dentata
A new method for the measurement of endogenous transmitter release in localized regions of hippocampal slices
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