Soraia Melo
0000-0002-0278-0827
Universidade do Porto Instituto de Investigação e Inovação em Saúde
48 papers found
Refreshing results…
E-cadherin variants associated with oral facial clefts trigger aberrant cell motility in a REG1A-dependent manner
The ECM and tissue architecture are major determinants of early invasion mediated by E-cadherin dysfunction
Physical and biochemical nuclear signatures: a secret message underlying gastric cancer invasion
Unraveling new strategies for early detection of invasive cancer cells: A special focus on the nuclear and cytoskeletal architecture
Nucleus positioning and composition dictate the invasion performance of E-cadherin mutant cells
Revisiting the Biological and Clinical Impact of CDH1 Missense Variants
Variantes da Caderina-E associadas a espectros clínicos distintos induzem diferentes perfis de migração e de adesão celular
Motile and adhesive profiles of variants associated with distinct E-cadherin mediated disorders
Cell extrusion: a synergistic interaction between E- cadherin and Filamin A
Nuclear signatures of an invasive cancer program triggered by E-cadherin dysfunction
Unravelling New Strategies for Early Detection of Invasive Cancer Cells: a Focus on the Nuclear Architecture Dynamics
Filamin A is critical regulator of the extrusive abilities mediated by E-cadherin dysfunction
E-cadherin mutations induce remodeling of nucleus positioning and composition towards an invasive cancer program
E-cadherin variants associated with distinct clinical phenotypes induce distinct motile and adhesive profiles
Motile and adhesive signatures of E-cadherin variants associated with distinct clinical phenotypes
Germline CDH1 G212E Missense Variant: Combining Clinical, In Vitro and In Vivo Strategies to Unravel Disease Burden
Diagnosis of cancer-causative E-cadherin missense mutations through molecular and morphological changes involved in cell extrusion
Unraveling the molecular and morphological features involved in basal extrusion of E-cadherin dysfunctional cells
Cell-ECM adhesion features determine the clinical spectrum of E-cadherin germline mutations
The Extracellular Matrix: An Accomplice in Gastric Cancer Development and Progression
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