Davide Lucchesi
0000-0002-8019-556X
Kymab Ltd
48 papers found
Refreshing results…
T follicular-helper cells (TFH) enrichment and follicular T regulatory cells (TFR) exclusion from ectopic germinal centers in salivary glands of Sjogren's Syndrome patients
OP0300 Enrichment of T follicular-helper cells (TFH) and exclusion of t follicular-regulatory cells (TFR) from ectoPIC germinal centers in salivary glands of sjogren's syndrome patients
01.09 Myeloid cells drive early inflammation and orchestrate salivary glands tertiary lymphoid structures formation in a model of inducible sialadenitis
Spontaneous and Inducible Animal Models of Sjögren’s Syndrome
IL-22 regulates lymphoid chemokine production and assembly of tertiary lymphoid organs
AB0169 Recruitment and Activation of NK Cells in the Salivary Glands Regulates Early Viral Control but is Dependable for Autoimmunity and Focal Lymphocytic Sialoadenitis in an Inducible Murine Model of Sjogren-Like Disease
Development and description of a novel inducible model of salivary gland inflammation in C57BL/6 mice characterised by tertiary lymphoid structures, autoimmunity and exocrine dysfunction
Recruitment and activation of NK cells in murine salivary glands regulate early immune response to viral infection but are dispensable for inducible lymphoneogenesis and autoimmunity
EBV and other viruses as triggers of tertiary lymphoid structures in primary Sjögren’s syndrome
Transcriptomic Profiling of the Development of the Inflammatory Response in Human Monocytes In Vitro
Innate immune system early activation precedes the formation of functional tertiary lymphoid structures in a novel model of salivary gland ectopic lymphoid neogenesis: 262
Transcriptomic profiling of the development of the inflammatory response in human monocytes in vitro e87680
Recruitment and activation of NK cells regulates early immune responses to viral infection in the salivary glands of an inducible murine model of Sjogren-like disease: 221
The role of viruses in autoreactive B cell activation within tertiary lymphoid structures in autoimmune diseases
A6. 2 Inducible Tertiary Lymphoid Structures and Autoimmunity in a Novel model of Sialoadenitis in Wild-Type Mice
Inducible tertiary lymphoid structures, autoimmunity, and exocrine dysfunction in a novel model of salivary gland inflammation in C57BL/6 mice
Inducible tertiary lymphoid structures formation in a novel model of sialoadenitis in wild-type mice is preceded by ectopic expression of lymphoid chemokines: P0013
Galectin-3 binds N eisseria meningitidis and increases interaction with phagocytic cells
Galectin-3 binds Neisseria meningitidis and increases interaction with phagocytic cells
Problems and challenges in the development and validation of human cell-based assays to determine nanoparticle-induced immunomodulatory effects
Missing publications? Search for publications with a matching author name.