Thomas Krieg
0000-0002-5192-580X
University of Cambridge
91 papers found
Refreshing results…
NECA at reperfusion limits infarction and inhibits formation of the mitochondrial permeability transition pore by activating p70S6 kinase
Effects of antibodies obtained from patients with dilated cardiomyopathy on the function of isolated rat hearts
Peptide blockers of PKG inhibit ROS generation by acetylcholine and bradykinin in cardiomyocytes but fail to block protection in the whole heart
Multiple, brief coronary occlusions during early reperfusion protect rabbit hearts by targeting cell signaling pathways
Acetylcholine and bradykinin trigger preconditioning in the heart through a pathway that includes Akt and NOS
Mitochondrial ROS generation following acetylcholine-induced EGF receptor transactivation requires metalloproteinase cleavage of proHB-EGF
Augmented taurine release is not the mechanism of ischemic preconditioning?s cardioprotection
Bradykinin induces mitochondrial ROS generation via NO, cGMP, PKG, and mitoKATPchannel opening and leads to cardioprotection
Mitochondria and their role in preconditioning's trigger phase
Activation of Akt is essential for acetylcholine to trigger generation of oxygen free radicals
ACh and adenosine activate PI3-kinase in rabbit hearts through transactivation of receptor tyrosine kinases
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