Miriam Fichtner
0000-0002-0107-4103
Yale University
8 papers found
Refreshing results…
The mechanisms of immunopathology underlying B cell depletion therapy-mediated remission and relapse in patients with MuSK MG
Heterogeneity of Acetylcholine Receptor Autoantibody–Mediated Complement Activity in Patients With Myasthenia Gravis
Myasthenia gravis complement activity is independent of autoantibody titer and disease severity
Elevated N-Linked Glycosylation of IgG V Regions in Myasthenia Gravis Disease Subtypes
Complement activity in myasthenia gravis is independent of autoantibody titer and disease severity
Affinity maturation is required for pathogenic monovalent IgG4 autoantibody development in myasthenia gravis
Single-cell repertoire tracing identifies rituximab-resistant B cells during myasthenia gravis relapses
Self-antigen driven affinity maturation is required for pathogenic monovalent IgG4 autoantibody development
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