Markus Gräler
0000-0001-6650-7849
79 papers found
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The role of sphingosine-1-phosphate signaling in HSV-1-infected human umbilical vein endothelial cells
A therapy with miglustat, 2-hydroxypropyl-ß-cyclodextrin and allopregnanolone restores splenic cholesterol homeostasis in Niemann-pick disease type C1
Neural sphingosine 1-phosphate accumulation activates microglia and links impaired autophagy and inflammation
Loss of sphingosine 1-phosphate (S1P) in septic shock is predominantly caused by decreased levels of high-density lipoproteins (HDL)
Regulation of ABCA1-mediated cholesterol efflux by sphingosine-1-phosphate signaling in macrophages
Acid Sphingomyelinase Inhibition Stabilizes Hepatic Ceramide Content and Improves Hepatic Biotransformation Capacity in a Murine Model of Polymicrobial Sepsis
Potent anti-inflammatory properties of HDL in vascular smooth muscle cells mediated by HDL-S1P and their impairment in coronary artery disease due to lower HDL-S1P: a new aspect of HDL dysfunction and its therapy
Influence of sphingosine-1-phosphate signaling on HCMV replication in human embryonal lung fibroblasts
Targeting sphingosine-1-phosphate lyase as an anabolic therapy for bone loss
Release of Platelet-Derived Sphingosine-1-Phosphate Involves Multidrug Resistance Protein 4 (MRP4/ABCC4) and Is Inhibited by Statins
Hepatocyte nuclear factor 1A deficiency causes hemolytic anemia in mice by altering erythrocyte sphingolipid homeostasis
Acid Sphingomyelinase Inhibition Prevents Development of Sepsis Sequelae in the Murine Liver
Plasma sphingosine-1-phosphate concentrations are associated with systolic heart failure in patients with ischemic heart disease
Sphingosine-1-Phosphate
Adjustment of Dysregulated Ceramide Metabolism in a Murine Model of Sepsis-Induced Cardiac Dysfunction
SGPL1 (sphingosine phosphate lyase 1) modulates neuronal autophagy via phosphatidylethanolamine production
Ceramide synthase 2 facilitates S1P-dependent egress of thymocytes into the circulation in mice
Tumor specific regulatory T cells in the bone marrow of breast cancer patients selectively upregulate the emigration receptor S1P1
Sphingosine 1-phosphate lyase ablation disrupts presynaptic architecture and function via an ubiquitin- proteasome mediated mechanism
Modulating sphingosine 1-phosphate signaling with DOP or FTY720 alleviates vascular and immune defects in mouse sepsis
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