Feng Li
0000-0001-5868-7456
China Medical University
28 papers found
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Histone demethylase Jumonji domain-containing 1A inhibits proliferation and progression of gastric cancer by upregulating runt-related transcription factor 3.
Histone demethylase JMJD1A inhibits proliferation and progression of gastric cancer by up‐regulating RUNX3
Mitochondrial PAK6 inhibits prostate cancer cell apoptosis via the PAK6-SIRT4-ANT2 complex
A novel PAK4-CEBPB-CLDN4 axis involving in breast cancer cell migration and invasion
MORC2 promotes development of an aggressive colorectal cancer phenotype through inhibition of NDRG1
A mandatory role of nuclear PAK4-LIFR axis in breast-to-bone metastasis of ERα-positive breast cancer cells
Exosomes from BM-MSCs increase the population of CSCs via transfer of miR-142-3p
HSF1, in association with MORC2, downregulates ArgBP2 via the PRC2 family in gastric cancer cells
Structure-Based Design of 6-Chloro-4-aminoquinazoline-2-carboxamide Derivatives as Potent and Selective p21-Activated Kinase 4 (PAK4) Inhibitors
A Novel Pak1/ATF2/miR-132 Signaling Axis Is Involved in the Hematogenous Metastasis of Gastric Cancer Cells
LC-0882 targets PAK4 and inhibits PAK4-related signaling pathways to suppress the proliferation and invasion of gastric cancer cells.
PAK4 regulates G6PD activity by p53 degradation involving colon cancer cell growth
Structure, biochemistry, and biology of PAK kinases.
GL-1196 Suppresses the Proliferation and Invasion of Gastric Cancer Cells via Targeting PAK4 and Inhibiting PAK4-Mediated Signaling Pathways
Group II p21-activated kinases as therapeutic targets in gastrointestinal cancer
Upregulated LMO1 in prostate cancer acts as a novel coactivator of the androgen receptor
PAK5-mediated E47 phosphorylation promotes epithelial–mesenchymal transition and metastasis of colon cancer
CREB is a key negative regulator of carbonic anhydrase IX (CA9) in gastric cancer
PAK1-mediated MORC2 phosphorylation promotes gastric tumorigenesis
GATA1 induces epithelial-mesenchymal transition in breast cancer cells through PAK5 oncogenic signaling
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