Ji Bihl
0000-0003-2404-1782
Wright State University
22 papers found
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Thermal Burn Injury Generates Bioactive Microvesicles: Evidence for a Novel Transport Mechanism for the Lipid Mediator Platelet-Activating Factor (PAF) That Involves Subcellular Particles and the PAF Receptor
ACE2-EPC-EXs protect ageing ECs against hypoxia/reoxygenation-induced injury through the miR-18a/Nox2/ROS pathway
Glioma stem cells-derived exosomes promote the angiogenic ability of endothelial cells through miR-21/VEGF signal
UVB-generated Microvesicle Particles: A Novel Pathway by Which a Skin-specific Stimulus Could Exert Systemic Effects
NPC-EXs Alleviate Endothelial Oxidative Stress and Dysfunction through the miR-210 Downstream Nox2 and VEGFR2 Pathways
Analyses of Endothelial Cells and Endothelial Progenitor Cells Released Microvesicles by Using Microbead and Q-Dot Based Nanoparticle Tracking Analysis
The effects of microvesicles on endothelial progenitor cells are compromised in type 2 diabetic patients via downregulation of the miR-126/VEGFR2 pathway
Endothelial progenitor cells and neural progenitor cells synergistically protect cerebral endothelial cells from Hypoxia/reoxygenation-induced injury via activating the PI3K/Akt pathway
UVB Generates Microvesicle Particle Release in Part Due to Platelet-activating Factor Signaling
Microvascular endothelial cells-derived microvesicles imply in ischemic stroke by modulating astrocyte and blood brain barrier function and cerebral blood flow
The Novel Methods for Analysis of Exosomes Released from Endothelial Cells and Endothelial Progenitor Cells
Angiotensin-(1–7) counteracts the effects of Ang II on vascular smooth muscle cells, vascular remodeling and hemorrhagic stroke: Role of the NFкB inflammatory pathway
The Role of Circulating Platelets Microparticles and Platelet Parameters in Acute Ischemic Stroke Patients
Angiotensin-(1–7) counteracts angiotensin II-induced dysfunction in cerebral endothelial cells via modulating Nox2/ROS and PI3K/NO pathways
Cellular Membrane Microparticles: Potential Targets of Combinational Therapy for Vascular Disease
Activation of the ACE2/Ang-(1–7)/Mas pathway reduces oxygen–glucose deprivation-induced tissue swelling, ROS production, and cell death in mouse brain with angiotensin II overproduction
The Preliminary Study of Effects of Tolfenamic Acid on Cell Proliferation, Cell Apoptosis, and Intracellular Collagen Deposition in Keloid FibroblastsIn Vitro
Proteomic analysis of circulating microparticles from db/db diabetic mice
Second International Meeting of ISEV 2013: Boston, USA, April 17th-20th, 2013.
Circulating microparticles from db/db diabetic mice induce dysfunction and apoptosis to cerebral endothelial cells
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