Michael Gold
0000-0002-2083-6206
University of Pittsburgh
78 papers found
Refreshing results…
Cutaneous and Colonic Rat DRG Neurons Differ With Respect to Both Baseline and PGE2-Induced Changes in Passive and Active Electrophysiological Properties
Characterization of basal and re-inflammation-associated long-term alteration in pain responsivity following short-lasting neonatal local inflamatory insult:
Spinal nerve injury increases the percentage of cold-responsive DRG neurons
Cold transduction in rat trigeminal ganglia neurons in vitro
TRPM8 mRNA Is Expressed in a Subset of Cold-Responsive Trigeminal Neurons From Rat
Changes in TrkB-like Immunoreactivity in Rat Trigeminal Ganglion After Tooth Injury
Inhibition of neuropathic pain by decreased expression of the tetrodotoxin-resistant sodium channel, NaV1.8:
Mechanisms of pain
Spinal nerve ligation: what to blame for the pain and why:
Ion Channels of Nociception.
A tetrodotoxin-resistant sodium current mediates inflammatory pain in the rat
Immobility and flight associated with antinociception produced by activation of the ventral and lateral/dorsal regions of the rat periaqueductal gray
DAMGO inhibits prostaglandin E2-induced potentiation of a TTX-resistant Na+ current in rat sensory neurons in vitro
Co-expression of nociceptor properties in dorsal root ganglion neurons from the adult rat in vitro
Role of a Ca2+-dependent slow afterhyperpolarization in prostaglandin E2-induced sensitization of cultured rat sensory neurons
Hyperalgesic agents increase a tetrodotoxin-resistant Na+ current in nociceptors.
Catecholamine-induced mechanical sensitization of cutaneous nociceptors in the rat
Periaqueductal gray stimulation produces a spinally mediated, opioid antinociception for the inflamed hindpaw of the rat
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