54 papers found
N-Glycosylation Regulates Pannexin 2 Localization but Is Not Required for Interacting with Pannexin 1
Erratum to: Inhibition of pannexin1 channels alleviates acetaminophen-induced hepatotoxicity
Inhibition of pannexin1 channels alleviates acetaminophen-induced hepatotoxicity
Connexin and pannexin channels in cancer
Loss of Panx1 Impairs Mammary Gland Development at Lactation: Implications for Breast Tumorigenesis
Pannexin 3, but not pannexin 1 is an important pro-catabolic mediator in osteoarthritis.
Inhibition of PANX1 reduces the malignant properties of human melanoma.
Lack of N-glycosylation of Pannexin 2 triggers aggregation in the endoplasmic reticulum without ablating its capacity to traffic to the cell surface.
Pannexin 1 regulates adipocyte development and fat accumulation
Deletion of Panx3 Prevents the Development of Surgically Induced Osteoarthritis
The Role of Pannexins in Fat Accumulation and Metabolism
Inhibition of Pannexin1 Channels Alleviates Acetaminophen-Induced Hepatotoxicity in Mouse
Phenotypic Analysis of Long Bones in Pannexin 3 Knockout Mice Using a Geometric Morphometric Approach
Abstract 2467: Connexin43 reduces melanoma growth within a keratinocyte microenvironment and during tumorigenesisin vivo
Pannexin 1 and Pannexin 3 Channels Regulate Skeletal Muscle Myoblast Proliferation and Differentiation*
Chemotherapeutic Drugs Induce ATP Release via Caspase-gated Pannexin-1 Channels and a Caspase/Pannexin-1-Independent Mechanism.
Pannexin channels and their links to human disease
Pannexin 3: A new channel into the mechanisms of osteoarthritis
Panx1 Regulates Cellular Properties of Keratinocytes and Dermal Fibroblasts in Skin Development and Wound Healing
Regulation of pannexin channels by post-translational modifications
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