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Intestinal tuft cell immune privilege enables norovirus persistence
UploadIFN-λ derived from nonsusceptible enterocytes acts on tuft cells to limit persistent norovirus
Download from doi.orgPLSCR1 is a cell-autonomous defence factor against SARS-CoV-2 infection
UploadDYRK1A promotes viral entry of highly pathogenic human coronaviruses in a kinase-independent manner
Download from doi.orgPharmacological disruption of mSWI/SNF complex activity restricts SARS-CoV-2 infection
UploadTuft-cell-intrinsic and -extrinsic mediators of norovirus tropism regulate viral immunity
Download from doi.orgOmicron-specific mRNA vaccination alone and as a heterologous booster against SARS-CoV-2
Download from www.nature.comInflammasome activation in infected macrophages drives COVID-19 pathology
Download from www.nature.comTuft cells are key mediators of interkingdom interactions at mucosal barrier surfaces
Download from doi.orgA humanized mouse model of chronic COVID-19
UploadHigh-affinity, neutralizing antibodies to SARS-CoV-2 can be made without T follicular helper cells.
UploadRestriction of SARS-CoV-2 replication by targeting programmed −1 ribosomal frameshifting
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