Guo-Min Li
0000-0002-9842-4578
The University of Texas Southwestern Medical Center
8 papers found
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Mutant huntingtin protein induces MLH1 degradation, DNA hyperexcision, and cGAS–STING-dependent apoptosis
DNA mismatch repair in cancer immunotherapy
The mismatch recognition protein MutSα promotes nascent strand degradation at stalled replication forks
DNA-PKcs-dependent phosphorylation of RECQL4 promotes NHEJ by stabilizing the NHEJ machinery at DNA double-strand breaks
Mispair-bound human MutS–MutL complex triggers DNA incisions and activates mismatch repair
HDAC3 deacetylates the DNA mismatch repair factor MutSβ to stimulate triplet repeat expansions
A PoleP286R mouse model of endometrial cancer recapitulates high mutational burden and immunotherapy response
Phosphorylation of proliferating cell nuclear antigen promotes cancer progression by activating the ATM/Akt/GSK3β/Snail signaling pathway
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