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Wiley, Clinical Endocrinology, 1(76), p. 72-77, 2011

DOI: 10.1111/j.1365-2265.2011.04161.x

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Multiple forms of hypogonadism of central, peripheral or combined origin in males with Prader-Willi syndrome

Journal article published in 2011 by Antonio F. Radicioni, G. Di Giorgio, G. Grugni, M. Cuttini, V. Losacco, A. Anzuini, S. Spera, C. Marzano, Andrea Lenzi, M. Cappa, A. Crino ORCID
This paper was not found in any repository, but could be made available legally by the author.
This paper was not found in any repository, but could be made available legally by the author.

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Abstract

BACKGROUND: Hypogonadism in Prader-Willi syndrome (PWS) is generally attributed to hypothalamic dysfunction or to primary gonadal defect, but pathophysiology is still unclear. OBJECTIVES: To investigate the aetiology of hypothalamic-pituitary-gonadal axis dysfunction in PWS males. METHODS: Clinical examination and blood sampling for luteinizing hormone (LH), follicle-stimulating hormone (FSH), testosterone, inhibin B and sexhormone-binding globulin (SHBG) were performed in 34 PWS patients, age 5·1-42·7 years, and in 125 healthy males of same age range. All participants were divided into two groups :