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American Society of Hematology, Blood, 7(124), p. 992-993, 2014

DOI: 10.1182/blood-2014-07-583856

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Is JAK2V617F finally off the hook?

Journal article published in 2014 by Ingeborg Hers ORCID, Alastair W. Poole ORCID
This paper is made freely available by the publisher.
This paper is made freely available by the publisher.

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Abstract

In this issue of Blood, Lamrani et al use various mouse models to demonstrate that the disease phenotype, rather than the JAK2(V617F) mutation, is responsible for the increased risk of thrombosis and bleeding in mouse models of myeloproliferative neoplasms (MPNs).