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Portland Press, Biochemical Society Transactions, 1(35), p. 133-135, 2007

DOI: 10.1042/bst0350133

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Differential role of TRP channels in prostate cancer.

Journal article published in 2007 by Natacha Prevarskaya, Matthieu Flourakis ORCID, Gabriel Bidaux, Stéphanie C. Thebault, Roman Skryma
This paper is available in a repository.
This paper is available in a repository.

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Abstract

A major clinical problem with PC (prostate cancer) is the cell's ability to survive and proliferate upon androgen withdrawal. Indeed, deregulated cell differentiation and proliferation, together with the suppression of apoptosis, provides the condition for abnormal tissue growth. Here, we examine the differential role of TRP (transient receptor potential) channels in the control of Ca(2+) homoeostasis and growth of PC cells.