Dissemin is shutting down on January 1st, 2025

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Wiley, Immunological Reviews, 1(219), p. 103-117, 2007

DOI: 10.1111/j.1600-065x.2007.00544.x

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To activate or not to activate: Distinct strategies used by Helicobacter pylori and Francisella tularensis to modulate the NADPH oxidase and survive in human neutrophils

Journal article published in 2007 by Lee-Ann H. Allen ORCID, Lee‐Ann H. Allen, Ramona L. McCaffrey
This paper is available in a repository.
This paper is available in a repository.

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Abstract

Neutrophils accumulate rapidly at sites of infection, and the ability of these cells to phagocytose and kill microorganisms is an essential component of the innate immune response. Relatively few microbial pathogens are able to evade neutrophil killing. Herein, we describe the novel strategies used by Helicobacter pylori and Francisella tularensis to disrupt neutrophil function, with a focus on assembly and activation of the nicotinamide adenine dinucleotide phosphate (NADPH) oxidase.