Elsevier, Neuroscience, (300), p. 576-584, 2015
DOI: 10.1016/j.neuroscience.2015.05.048
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•We investigate the role of PPARγ and PPARδ in a model of Parkinson’s disease.•Conditional neuronal knockout of PPARγ or PPARδ did not increase susceptibility to MPTP.•Conditional double knockout of neuronal PPARγ and PPARδ did not enhance MPTP toxicity significantly.•Glial PPAR expression seems more important to confer neuroprotection.