American Physiological Society, AJP - Endocrinology and Metabolism, 1(323), p. E53-E68, 2022
DOI: 10.1152/ajpendo.00001.2022
Full text: Unavailable
New molecular mechanisms are important to unravel and understand diabetic polyneuropathy, a disorder prevalent in over half of persons with diabetes mellitus (DM). MUPs, members of the lipocalin family of molecules, have an unexpected impact on the plasticity of sensory neurons that are targeted in type 1 experimental diabetic neuropathy. This work explores this potential target in neuropathy in the context of the lipocalin family of molecules.