Dissemin is shutting down on January 1st, 2025

Published in

American Society for Cell Biology, Molecular Biology of the Cell, 14(33), 2022

DOI: 10.1091/mbc.e21-10-0499

Links

Tools

Export citation

Search in Google Scholar

The [PSI<sup>+</sup>] prion modulates cytochrome c oxidase deficiency caused by deletion of COX12

This paper was not found in any repository, but could be made available legally by the author.
This paper was not found in any repository, but could be made available legally by the author.

Full text: Unavailable

Red circle
Preprint: archiving forbidden
Green circle
Postprint: archiving allowed
Orange circle
Published version: archiving restricted
Data provided by SHERPA/RoMEO

Abstract

Yeast cells lacking the Cox12 subunit of Cytochrome c (CcO) oxidase are impaired in mitochondrial respiration. With a combination of experimental evolution, genetics, and biochemistry, we prove that elimination of the PSI+ prion improves CcO oxidase activity and respiratory metabolism in strains lacking Cox12.