American Physiological Society, American Journal of Physiology - Lung Cellular and Molecular Physiology, 5(325), p. L675-L688, 2023
DOI: 10.1152/ajplung.00281.2022
Full text: Unavailable
In contrast to the abundant literature of mechanical pulmonary artery clamping to increase right ventricular afterload, we developed a model adding a biological factor of pulmonary ischemia-reperfusion injury. We did not only focus on the right ventricular behavior, but also on the interaction with the injured lung. We are the first to describe this interaction while addressing the hemodynamic intraoperative phases of clinical lung transplantation.