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American Association for the Advancement of Science, Science Translational Medicine, 605(13), 2021

DOI: 10.1126/scitranslmed.abe0407

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TGFβ2 and TGFβ3 isoforms drive fibrotic disease pathogenesis

This paper was not found in any repository, but could be made available legally by the author.
This paper was not found in any repository, but could be made available legally by the author.

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Abstract

TGFβ2 and TGFβ3 isoforms are activated via unique mechanisms and are associated with fibrosis in human diseases.