American Association for the Advancement of Science, Science Translational Medicine, 529(12), 2020
DOI: 10.1126/scitranslmed.aay1809
Full text: Unavailable
APOE4 exacerbates α-synuclein pathology in a synucleinopathy mouse model, and APOE4 genotype associates with increased α-synuclein pathology in humans.