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American Association for the Advancement of Science, Science Translational Medicine, 413(9), 2017

DOI: 10.1126/scitranslmed.aaj2347

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Transplantation of wild-type mouse hematopoietic stem and progenitor cells ameliorates deficits in a mouse model of Friedreich’s ataxia

This paper is available in a repository.
This paper is available in a repository.

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Abstract

YG8R mice, a model of Friedreich’s ataxia, show amelioration of the disease phenotype when transplanted with wild-type mouse hematopoietic stem and progenitor cells.