244 papers found
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Functional Interactions between Retinoblastoma and c-MYC in a Mouse Model of Hepatocellular Carcinoma
Abstract 2384: A novel mouse model for the study of c-MYC during initiation and progression of pancreatic ductal adenocarcinoma
Abstract 2069: Regulation of nucleostemin by reactive oxygen species
MYC Phosphorylation, Activation, and Tumorigenic Potential in Hepatocellular Carcinoma are Regulated by HMG-CoA Reductase
Reactive Oxygen Species Regulate Nucleostemin Oligomerization and Protein Degradation*
Real-time quantitative PCR of: (A) human c-MYC mRNA, (B) mouse Ornithine Decarboxylase (ODC) mRNA, (C) mouse Nucleolin mRNA, and (D) mouse Cyclin B1 mRNA
(A) Immunofluorescence for Ki67 expression as a marker of liver cell proliferation is presented
(A) Kaplan-Meir analysis of Tumor Free Survival for oncogene-induced lung tumorigenesis
Shown are Kaplan-Meier survival curves for 6–8 w adult mice that overexpressed MYC and were simultaneously treated with: nothing (filled black square), DDC (filled blue circle), or CCl (filled blue diamond)
ABT-737 Targets Leukemic Stem Cells In Mouse Models of Mutant NRASD12/hBCL-2- Mediated Acute Myeloid Leukemia Progression with Increased Survival
CD4+ T-cells Contribute to the Remodeling of the Microenvironment Required for Sustained Tumor Regression upon Oncogene Inactivation
Definition of an enhanced immune cell therapy in mice that can target stem-like lymphoma cells
Nanoscale approaches to define biologic signatures and measure proteomic response to targeted therapies in hematologic and solid tumors
Development of a MicroCT-Based Image-Guided Conformal Radiotherapy System for Small Animals
Noninvasive molecular imaging of c-Myc activation in living mice
Impact of Hydrodynamic Injection and phiC31 Integrase on Tumor Latency in a Mouse Model of MYC-Induced Hepatocellular Carcinoma
The interaction between Myc and Miz1 is required to antagonize TGFβ-dependent autocrine signaling during lymphoma formation and maintenance
MYC Inactivation Elicits Oncogene Addiction through Both Tumor Cell–Intrinsic and Host-Dependent Mechanisms
Conditional TPM3-ALK and NPM-ALK transgenic mice develop reversible ALK-positive early B-cell lymphoma/leukemia.
Abstract 5070: Identification of DKK2 as a candidate gene that facilitates tumor reoccurrence upon MYC inactivation
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