87 papers found
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HIPK2 role in the tumor–host interaction: Impact on fibroblasts transdifferentiation CAF‐like
Reduced chemotherapeutic sensitivity in high glucose condition: implication of antioxidant response
Autophagy manipulation as a strategy for efficient anticancer therapies: possible consequences
Mutant p53, Stabilized by Its Interplay with HSP90, Activates a Positive Feed-Back Loop Between NRF2 and p62 that Induces Chemo-Resistance to Apigenin in Pancreatic Cancer Cells
Mutant p53 and Cellular Stress Pathways: A Criminal Alliance That Promotes Cancer Progression
STAT3 phosphorylation affects p53/p21 axis and KSHV lytic cycle activation
Could autophagy dysregulation link neurotropic viruses to Alzheimer’s disease?
Impact of HHV-6A and HHV-6B lytic infection on autophagy and endoplasmic reticulum stress
Cytotoxic Drugs Activate KSHV Lytic Cycle in Latently Infected PEL Cells by Inducing a Moderate ROS Increase Controlled by HSF1, NRF2 and p62/SQSTM1
EBV and KSHV Infection Dysregulates Autophagy to Optimize Viral Replication, Prevent Immune Recognition and Promote Tumorigenesis
EBV up-regulates PD-L1 on the surface of primary monocytes by increasing ROS and activating TLR signaling and STAT3
Metformin triggers apoptosis in PEL cells and alters bortezomib-induced Unfolded Protein Response increasing its cytotoxicity and inhibiting KSHV lytic cycle activation
Apigenin, by activating p53 and inhibiting STAT3, modulates the balance between pro-apoptotic and pro-survival pathways to induce PEL cell death
Oxidant species are involved in T/B-mediated ERK1/2 phosphorylation that activates p53-p21 axis to promote KSHV lytic cycle in PEL cells
Bortezomib promotes KHSV and EBV lytic cycle by activating JNK and autophagy
p53-Dependent PUMA to DRAM antagonistic interplay as a key molecular switch in cell-fate decision in normal/high glucose conditions
Histone deacetylase inhibitors VPA and TSA induce apoptosis and autophagy in pancreatic cancer cells
Quercetin induces apoptosis and autophagy in Primary Effusion Lymphoma cells by inhibiting PI3K/AKT/mTOR and STAT3 signaling pathways
Reactivation of mutant p53 by capsaicin, the major constituent of peppers
Immunogenic Cell Death in Cancer: From Benchside Research to Bedside Reality
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