Public Library of Science, PLoS ONE, 4(8), p. e60560, 2013
DOI: 10.1371/journal.pone.0060560
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Background: The role and mechanism of action of MIF in hyperoxia-induced acute lung injury (HALI) in the newborn lung are not known. We hypothesized that MIF is a critical regulatory molecule in HALI in the developing lung.