Jianmin Zuo
0000-0002-8341-465X
The University of Birmingham
46 papers found
Refreshing results…
ULBPs
NK cells produce high levels of IL-10 early after allogeneic stem cell transplantation and suppress development of acute GVHD
The number of CD56dim NK cells in the graft has a major impact on risk of disease relapse following allo-HSCT
A disease-linked ULBP6 polymorphism inhibits NKG2D-mediated target cell killing by enhancing the stability of NKG2D ligand binding
The Transcription Factor Hobit Identifies Human Cytotoxic CD4+ T Cells
NK cell function is markedly impaired in patients with chronic lymphocytic leukaemia but is preserved in patients with small lymphocytic lymphoma
Cytomegalovirus Infection Leads to Development of High Frequencies of Cytotoxic Virus-Specific CD4+ T Cells Targeted to Vascular Endothelium
Cytomegalovirus viral load within blood increases markedly in healthy people over the age of 70 years
Induction of lytic cycle sensitizes Epstein-Barr virus infected B cells to NK cell killing that is counteracted by virus-mediated NK cell evasion mechanisms in late lytic cycle
Identification of Epstein-Barr Virus Replication Proteins in Burkitt’s Lymphoma Cells
The missing link in EBV immune evasion
The Cellular Localization of Human Cytomegalovirus Glycoprotein Expression Greatly Influences the Frequency and Functional Phenotype of Specific CD4+ T Cell Responses
Epstein-Barr virus transcription factor Zta acts through distal regulatory elements to directly control cellular gene expression
Cooperation between Epstein-Barr Virus Immune Evasion Proteins Spreads Protection from CD8+ T Cell Recognition across All Three Phases of the Lytic Cycle
Robust T-cell stimulation by Epstein-Barr virus–transformed B cells after antigen targeting to DEC-205
Kaposi's Sarcoma-Associated Herpesvirus-Encoded Viral IRF3 Modulates Major Histocompatibility Complex Class II (MHC-II) Antigen Presentation through MHC-II Transactivator-Dependent and -Independent Mechanisms: Implications for Oncogenesis
Herpesviruses Placating the Unwilling Host: Manipulation of the MHC Class II Antigen Presentation Pathway
T-cell immunity to Kaposi's sarcoma-associated herpesvirus: recognition of primary effusion lymphoma with LANA-specific CD4+ T cells.
Epstein-Barr Virus Evades CD4+ T Cell Responses in Lytic Cycle through BZLF1-mediated Downregulation of CD74 and the Cooperation of vBcl-2
The Epstein-Barr virus-encoded BILF1 protein modulates immune recognition of endogenously processed antigen by targeting MHC class I molecules trafficking on both the exocytic and endocytic pathways.
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