Anna Nicolaou
www.research.manchester.ac.uk
0000-0002-1314-413X
The University of Manchester
210 papers found
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Three-way assessment of long-chain n-3 PUFA nutrition: by questionnaire and matched blood and skin samples.
Analysis of Prostamides E2 and F2a Produced in Rabbit Cornea.
Prostamides PGE2-EA and PGF2a-EA are found in rabbit cornea.
Prostanoids
Green tea catechins exhibit protective effects against UVR-induced skin inflammation in vivo and in relation to reduced levels of 12-HETE
Green tea catechins attenuate the production of 12-HETE in human skin following exposure to ultraviolet radiation.
The polyunsaturated fatty acids, EPA and DPA exert a protective effect in the hippocampus of the aged rat
Green tea catechins (GTC) are bioavailable in human skin and protect against UVR-induced skin inflammation.
Ultraviolet-radiation induced skin inflammation: dissecting the role of bioactive lipids
Lipidomics: What does the future hold?
Circulating COX-derived eicosanoids inhibit macrophage function in mice and humans with liver disease.
The immunomodulatory properties of mesenchymal stem cells and their use for immunotherapy
Mediator lipidomics reveal the role of eicosanoids in UVR-induced skin inflammation (Oral Presentation).
Lipidomic analysis reveals the effect of gamma linolenic acid treatment on C6 glioma cell eicosanoids (Oral Presentation).
Influence of dietary fish oil on the fatty acid composition of colonic epithelial cells.
The influence of n-3 LC-PUFA-supplemented yogurt on human erythrocyte fatty acid and plasma eicosanoid profiles.
High performance liquid chromatography assay for the detection of diphencyprone and the metabolite diphenylacetylene in serum and uring following topical application for the treatment of alopecial areata.
Exposure to commonly used surfactants induces alterations in the membrane fatty acid composition of staphylococci.
Temporal profiles show differential contribution of lipid mediators in uv-induced inflammation in sun- reactive skin types i and iv.
Biphasic response in prostanoid breakdown during UVR-induced inflammation of human skin.
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