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American Thoracic Society, American Journal of Respiratory and Critical Care Medicine, 8(180), p. 780-787, 2009

DOI: 10.1164/rccm.200810-1662oc

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Evidence of Dysfunction of Endothelial Progenitors in Pulmonary Arterial Hypertension

Journal article published in 2009 by Mark Toshner, Robert Voswinckel, Mark Southwood, Rafia Al-Lamki, Luke S. G. Howard, Denis Marchesan, Jun Yang, Jay Suntharalingam, Elaine Soon, Andrew Exley, Susan Stewart, Markus Hecker, Zhenping Zhu, Ursula Gehling, Werner Seeger and other authors.
This paper is available in a repository.
This paper is available in a repository.

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Abstract

Rationale: Severe pulmonary arterial hypertension (PAH) is characterized by the formation of plexiform lesions and concentric intimal fibrosis in small pulmonary arteries. The origin of cells contributing to these vascular lesions is uncertain. Endogenous endothelial progenitor cells are potential contributors to this process.